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Inflammation and its role in neuroprotection, axonal regeneration and functional recovery after spinal cord injury

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This can be genetically determined, but it can also be caused by inflammation of the intestinal mucosa (e.g

17-estradiol and naturally occurring plant-derived compounds like phytoestrogens found to have neuroprotective effects via activation of receptors like estrogen receptor- (ER), ER, or G-protein coupled receptor-1 (GPER1) and stimulation of the signaling cascades including (a) extracellular regulated kinase-1 (ERK1)/ERK2, (b) PI3K/Akt, and (c) c-Jun amino-terminal kinase (JNK), which regulate various downstream transcription factors implicated in the survival of the neurons [119, 120]

The regulation of diverse transcription factors, receptors for growth factors (including FGFR2b, FGFR2c, EGFR and HER2), and the activation of Akt are other elements in the reversion of MET [24]