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The core of this reprogramming lies in the alteration of membrane phospholipid composition, particularly an increased proportion of polyunsaturated fatty acids (PUFAs), thereby enhancing membrane lipid susceptibility to oxidative stress

Isik et al., 2023) Furthermore, excessive production of ROS has also been linked to dysfunctions in mitochondrial complex I and the complex activity reported to be reduced in PD patient brains (Keeney et al., 2006)

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In this study, we aim to explore whether chronic ethanol exposure leads to oxidative stress-mediated exacerbation of AD pathology, which in turn results in activation of microglia and neuroinflammatory damage, which ultimately leads to cognitive behavioral disorders

4.1 Interaction between fungal virulence factors and host mitochondria Fungal virulence factors directly target host mitochondria, profoundly disrupting bioenergetic and immune functions